Swine Diseases
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PRRS and Swine Influenza
Respiratory disease Nose- nose contact Cough, fever Labored breathing, nasal discharge Snout dis formation Dehydration Decrease feed and water consumption
APP& glasser’s disease
Respiratory disease Nose- nose contact Cough, fever Labored breathing, nasal discharge Snout dis formation Dehydration Decrease feed and water consumption
E. coli and salmonella
Enteric disease Nose to nose or nose to feces contact Diarrhea Dehydration Vomiting Lethargic Decrease water consumption Off feed
TGE and Swine Dysentery and coccidiosis
Enteric disease Nose to nose or nose to feces contact Diarrhea Dehydration Vomiting Lethargic Decrease water consumption Off feed
Strep. Suis& erysipelas & PMWS
Systematic disease Nose to nose contact or through air Poor condition Pale skin Swollen joints Convulsions Increases LN Decrease appetite
Swine pox&greasy pig disease&mange&lice
Skin diseases Poor condition Pale skin Dark color areas skin Flaking Decrease appetite
Transmissible gastroenteritis
Highly contagious VIRAL disease affecting pigs on all ages Newborns less than 10 Caused by corona virus Spread by aerosol, direct fecal contact or fomite Incubation
Transmissible gastroenteritis:host risk factors
Parity Herd immunity levels Environmental/management factors (all in all out)
Transmissible gastroenteritis: pathogen risk factors
Persists in environment for a few weeks Survives freezing Sensitive to light and disinfectants
Transmissible gastroenteritis: immune response
Strong antibody response ( IgA in intestinal tract protection) SIgA (excreted in sows milk and passive immunity for piglets) Best passive immunity from natural GI infection of the sow
Transmissible gastroenteritis: pathogenesis
Nasal secretions and milk-replicates respiratory intestinal tracts Destroys epithelial cells jejunum and Illium
Transmissible gastroenteritis: clinical signs
Vomiting, intense thirst, dehydration Diarrhea Pigs less than a year old die Abortion
Transmissible gastroenteritis: diagnosis
Clinical signs Necropsy Antibodies of intestinal contents
Transmissible gastroenteritis: treatment and control
No specific treatment Increase farrowing room temp Vax Feed ground up dead to sows Sanitation RECOVERED PIGS ARE IMMUNE FOR ONE YEAR
Atrophic Rhinitis & mycoplasmal pneumonia
Respiratory disease Nose- nose contact Cough, fever Labored breathing, nasal discharge Snout dis formation Dehydration Decrease feed and water consumption
E. Coli, salmonella, TGE, swine dysentery, coccidiosis
Enteric diseases(spread nose to nose to forced contact) Diarrhea Dehydration Vomiting Off feed Weird water amounts Lethargic
Strep. Suis, erysipelas, PMWS
Systematic diseases(nose to nose contact or through air) Poor condition, pale skin, dark areas on skin, swollen joints,
Skin diseases
Swine pox Greasy pig disease Mange Lice Pale skin Flaking
Transmissible gastroenteritis (TGE)
HIGHLY contagious a VIRAL disease Caused by coronavirus Spread through aerosol, direct fecal oral contact, or fomites Epidemic: explosive in naive herds–lasts 3-5 weeks Endemic: partial immunity, contagious introduction of susceptible pigs
Transmissible gastroenteritis (TGE): HOST risk factors
Parity Herd immunity levels Environmental/management factors (continuous flow)
Transmissible gastroenteritis (TGE):PATHOGEN risk factors
Persists in environments for a few weeks Survives freezing Sensitive to light aNd disinfectants
Immune response
Pigs develop strong antibody response (IgA) sIgA: excreted in Sows milk_passive immunity BEST PASSIVE IMMUNITY FROM NATURAL GI INFECTION OF SOW
Transmissible gastroenteritis (TGE): clinical signs
Vomiting, thirst, Diarrhea(yellow with bad odor) Abortion Subsequent infections not as severe
Transmissible gastroenteritis (TGE): diagnosis
Clinical signs Necropsy Fluorescent antibodies of intestinal contents